Agathe Genestar - Ostéopathe D.O générale et pédiatrique

Agathe Genestar - Ostéopathe D.O générale et pédiatrique Ostéopathe diplômée en juin 2023, j'ai par la suite enrichi mes connaissances par d'autres formations, notamment en pédiatrie.

21/06/2026
24/03/2026

𝗔 𝗗𝗲𝗲𝗽 𝗗𝗶𝘃𝗲 𝗶𝗻𝘁𝗼 𝗦𝗵𝗼𝘂𝗹𝗱𝗲𝗿 𝗠𝗲𝗰𝗵𝗮𝗻𝗶𝗰𝘀: 𝗛𝗼𝘄 𝗟𝗼𝗮𝗱 𝗗𝗶𝗿𝗲𝗰𝘁𝗶𝗼𝗻 𝗖𝗵𝗮𝗻𝗴𝗲𝘀 𝗦𝗰𝗮𝗽𝘂𝗹𝗮𝗿 𝗞𝗶𝗻𝗲𝗺𝗮𝘁𝗶𝗰𝘀

⬛ A recent 2025 study published in the Journal of Biomechanics by Lee et al. sheds new light on how our shoulder blades (scapulae) move depending on the specific mechanical demands of a task.
⬛ While we already know a lot about how the shoulder behaves when lifting an arm, this new research uncovers the fascinating mechanics behind pulling motions.

𝗧𝗵𝗲 𝗠𝗶𝘀𝘀𝗶𝗻𝗴 𝗟𝗶𝗻𝗸 𝗶𝗻 𝗦𝗵𝗼𝘂𝗹𝗱𝗲𝗿 𝗥𝗲𝘀𝗲𝗮𝗿𝗰𝗵

⬛ Historically, our understanding of healthy shoulder motion has been heavily based on studying the shoulder while it generates an abduction torque—essentially, raising or lowering the arm against gravity.
⬛ During arm raising, the deltoid muscle imposes a shear force that can destabilize the humeral head, so the scapula shifts to help stabilize the joint and prevent injury, such as impingement.
⬛ However, the human shoulder performs a wide variety of tasks beyond just raising our arms.
⬛ Specifically, very little attention has been given to concentric shoulder adduction (pulling the arms down against resistance).
⬛ This type of motion is critical for high-demand activities like rock climbing, vertical climbing, and wheelchair transfers.
⬛ Because shoulder adductors (like the latissimus dorsi and pectoralis major) are the prime movers during pulling tasks, they impose a completely different, inferior shear force on the joint compared to arm raising.

𝗛𝗼𝘄 𝘁𝗵𝗲 𝗦𝘁𝘂𝗱𝘆 𝗪𝗮𝘀 𝗖𝗼𝗻𝗱𝘂𝗰𝘁𝗲𝗱

⬛ To investigate these differences, the researchers compared a weighted press-up task (concentric abduction) to a weighted pull-down task (concentric adduction).

👥 Participants
⬛ The study recruited 10 young, healthy adults (5 males, 5 females) with no history of shoulder injuries.

🏋️ The Tasks
⬛ Participants performed a press-up with 7.5 kg of tension and a pull-down with 15 kg of tension using a controllable cable machine.
🎥 High-Tech Tracking
⬛ To track the intricate 3D movements of the bones, the researchers combined optical motion capture with biplanar videoradiography (XROMM).
⬛ This allowed them to capture highly accurate, direct measurements of the humerus, scapula, and thorax in motion.

𝗞𝗲𝘆 𝗙𝗶𝗻𝗱𝗶𝗻𝗴𝘀: 𝗧𝗵𝗲 𝗦𝗰𝗮𝗽𝘂𝗹𝗮 𝗶𝘀 𝗧𝗮𝘀𝗸-𝗦𝗽𝗲𝗰𝗶𝗳𝗶𝗰

⬛ The researchers predicted that even though press-ups and pull-downs look similar in terms of overall arm movement, the shoulder would achieve them using different internal mechanics.
⬛ Their findings strongly supported this:

📊 Greater Upward Rotation During Pull-Downs
⬛ The primary finding was that the scapula upwardly rotates significantly more during the pull-down task than the press-up task.
⬛ In fact, 9 out of 10 participants exhibited this distinct pattern.

🔄 Reduced Glenohumeral Abduction
⬛ Complementing the increased scapular rotation, there was significantly less glenohumeral (shoulder joint) abduction during the pulling motion compared to the pushing motion.

👤 High Individual Variability
⬛ The researchers also noted substantial inter-individual variability in how people moved their scapulae.
⬛ For instance, during the press-up task alone, the range of internal rotation varied across participants by approximately 30 degrees.

𝗪𝗵𝘆 𝗗𝗼𝗲𝘀 𝗧𝗵𝗶𝘀 𝗠𝗮𝘁𝘁𝗲𝗿? (𝗧𝗵𝗲 𝗕𝗶𝗼𝗺𝗲𝗰𝗵𝗮𝗻𝗶𝗰𝘀 𝗼𝗳 𝗦𝘁𝗮𝗯𝗶𝗹𝗶𝘁𝘆)

⬛ This study proves that scapula upward rotation is not simply a function of how high you lift your arm; it dynamically changes to accommodate the specific load experienced by the shoulder.
⬛ This load-specific adjustment likely exists to maximize joint stability.

⚙️ During a Pull-Down (Adduction)
⬛ The prime moving muscles pull the arm downward, creating an inferior (downward) destabilizing shear force.
⬛ By upwardly rotating the scapula more, the body naturally tilts the glenoid (the shoulder socket) more superiorly.
⬛ This re-orientation helps the muscles compress the joint together securely rather than shearing it apart.

⬆️ During a Press-Up (Abduction)
⬛ The deltoid pulls upward, creating a superior shear force.
⬛ The scapula rotates comparatively less, which keeps the glenoid oriented more inferiorly, effectively countering the deltoid's destabilizing pull.

𝗖𝗹𝗶𝗻𝗶𝗰𝗮𝗹 𝗧𝗮𝗸𝗲𝗮𝘄𝗮𝘆𝘀

⬛ This highly accurate kinematic data provides a much more well-rounded baseline for what constitutes "healthy" shoulder movement.
⬛ Often, clinicians look for "scapular dyskinesis" (abnormal shoulder blade movement) when treating shoulder pain.
⬛ By proving that healthy scapula motion naturally varies between pushing and pulling tasks, these findings can help physical therapists better identify true movement impairments in clinical populations and design more effective rehabilitation protocols.
⬛ Furthermore, the data will be instrumental in building better musculoskeletal computer models to simulate high-risk activities like rock climbing, potentially lowering injury rates in the future.

18/01/2026

Hot off the Press 🔥

𝗢𝘀𝘁𝗲𝗼𝗮𝗿𝘁𝗵𝗿𝗶𝘁𝗶𝘀 𝗮𝘀 𝗮 𝘀𝘆𝘀𝘁𝗲𝗺𝗶𝗰 𝗱𝗶𝘀𝗲𝗮𝘀𝗲

Historically, osteoarthritis (OA) has been narrowly understood as an unavoidable, cartilage-centric, wear-and-tear disease that is inevitable with ageing (https://pubmed.ncbi.nlm.nih.gov/22392533/). Growing evidence from both preclinical and clinical research, however, challenges this narrow perspective. Instead, OA is increasingly understood as a systemic condition that affects the whole person, shaped by ongoing interactions between joint tissues and biological processes throughout the body (https://pubmed.ncbi.nlm.nih.gov/41339496/).

⚖️Rather than being limited to mechanical damage, OA involves a combination of disrupted cellular metabolism, breakdown of the extracellular matrix, impaired repair mechanisms, and activation of innate immune pathways. These processes are not confined to the joint itself. They are strongly influenced by broader systemic factors, including ageing, obesity, sex-specific biology, metabolic disturbances, chronic inflammation, and inter-organ communication (https://pubmed.ncbi.nlm.nih.gov/30209413/, https://pubmed.ncbi.nlm.nih.gov/31621562/, figure 1 top). This broader view also helps explain a common clinical observation: structural joint damage seen on imaging often correlates poorly with pain severity. Pain in OA appears to be shaped not only by local tissue pathology but also by central and systemic mechanisms such as pain sensitization and psychosocial influences (https://pubmed.ncbi.nlm.nih.gov/31621573/, https://pubmed.ncbi.nlm.nih.gov/30307131/).

📘 A brand-new review by Collins and colleagues (https://pubmed.ncbi.nlm.nih.gov/41339496/) emphasizes the role of adipose tissue and metabolic dysfunction as active drivers of OA, rather than passive consequences of increased joint loading. Experimental studies show that adipokines, including leptin (a satiety hormone), as well as components of the complement system, can directly promote both joint damage and pain (https://pubmed.ncbi.nlm.nih.gov/32078923/, https://pmc.ncbi.nlm.nih.gov/articles/PMC5341385/). Notably, obesity-related OA can develop even in the absence of excessive mechanical stress on the joints (https://pubmed.ncbi.nlm.nih.gov/33443201/).

💪Loss of muscle mass and conditions such as sarcopenic obesity further increase the risk of OA and contribute to functional decline, highlighting the importance of body composition rather than body weight alone (https://pubmed.ncbi.nlm.nih.gov/23312414/, https://pubmed.ncbi.nlm.nih.gov/40746030/).

💊 The authors also stress the need to move beyond a one-size-fits-all view of OA by applying phenotyping and endotyping approaches. Identifying distinct biological subtypes of OA may help explain why disease-modifying osteoarthritis drugs (DMOADs) have repeatedly failed in clinical trials (https://pubmed.ncbi.nlm.nih.gov/41339496/). Factors such as heterogeneous patient populations, treatment initiation at advanced disease stages, and an overemphasis on single tissues or pathways have likely limited therapeutic success (https://pubmed.ncbi.nlm.nih.gov/29609224/). Encouragingly, systemically acting treatments—including GLP-1 receptor agonists, metformin, and anti-inflammatory drugs originally developed for metabolic or cardiovascular conditions—are emerging as promising options for specific OA subgroups (https://pubmed.ncbi.nlm.nih.gov/39476339/, https://pubmed.ncbi.nlm.nih.gov/40179178/).

🔄 In addition to the concept that systemic factors can influence OA, there is growing evidence that shows the effects of knee pain and damage on the rest of the organism, illustrating a bidirectional relationship. This concept has also been demonstrated in studies showing that knee injury and pain might cause changes to the cardiovascular system (https://www.oarsijournal.com/article/S1063-4584(24)00059-1/fulltext) , brain (https://pubmed.ncbi.nlm.nih.gov/36593507/, https://pubmed.ncbi.nlm.nih.gov/36374499/) adipose tissue (https://pubmed.ncbi.nlm.nih.gov/40279436/) and other organ systems (figure 1, bottom). These data suggest that systemic factors can drive OA and, in turn, OA can have systemic consequences (figure)). This bidirectional relationship reinforces the idea of OA as a chronic, multi-organ condition rather than an isolated joint disorder.

03/01/2026
Nouveau au cabinet à Flémalle!! La thérapie par Andullation! alliée de nombreuses autres thérapies, elle agit comme un b...
12/12/2025

Nouveau au cabinet à Flémalle!! La thérapie par Andullation! alliée de nombreuses autres thérapies, elle agit comme un boost métabolique. Prenez le temps d'y jeter un œil, vous pourriez aimer!

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