Movability - Wellness & Sport Sciences

Movability - Wellness & Sport Sciences Chiropractic, pelvic floor physiotherapy, acupuncture, massage, orthotics, naturopathic care, and rehab all under one roof.

We treat complex conditions through full-body assessment, root-cause care, and a collaborative, patient-first approach.

08/13/2026

If this makes your jaw relax instantly, congratulations: your nervous system just filed a screen-time complaint. 😂

What you did looks ridiculous, but the physiology is not.

Place the index and middle finger of one hand on either side of your nose, or use both index fingers. Keep your eyes fixed on the same target. This creates a crude form of binasal occlusion by blocking a sliver of the visual field beside your nose.

Your eyes still have to focus and converge on that target, so this is not simply “reducing convergence.” You did not stretch your masseter, mobilize your TMJ, or change tissue length. Yet some people will feel their jaw, temples, or upper neck relax within seconds.

That matters because tension is not only a property of a muscle. It is an output.

Your brain integrates visual input, eye position, vestibular signals, cervical proprioception, breathing, attention, threat, and task demand to decide how much stabilization to produce through the jaw, neck, and shoulders.

After hours at a screen, the system may be carrying more ocular effort, visual attention, near-focus demand, and postural fixation. In a susceptible person, that can spill into jaw bracing and cervical tone.

By masking part of the inner visual field, you may reduce competing visual information or provide a stable reference. We do not yet know the full mechanism. This does not mean the trick is “treating TMJ” or that your eyes are automatically the root cause.

Here is the part that should change how you think about pain:

If the tension changes before the tissue has time to change, the nervous system changed its decision.

This is how I investigate complex head, neck, jaw, dizziness, and post-concussion cases. I perturb one input, retest the output, and follow the physiology instead of assuming the painful structure is the whole problem.

Try it without intentionally unclenching. Comment JAW, TEMPLES, NECK, or NOTHING, and send this to someone glued to a laptop.

If it works instantly, the first prescription is still free: close the screen and go touch grass. 😂

Stop if it provokes dizziness, nausea, headache, or double vision.

Dr. Sina

08/10/2026

A neck MRI can look “mild” while the patient’s spinal cord is already struggling.

Degenerative cervical myelopathy happens when disc collapse, bone spurs, thickened ligaments, or a narrow spinal canal begin compressing and injuring the spinal cord.

The earliest signs are often not dramatic neck pain. They are small losses of function:

Dropping objects.
Buttons or handwriting becoming harder.
Numb, stiff, or clumsy hands.
Heavy legs.
Needing the railing on stairs.
Near falls.
Feeling less steady when turning or walking in the dark.

It gets missed because each symptom is explained separately.

The hands get labelled carpal tunnel.
The legs get blamed on the lower back.
The imbalance gets blamed on the inner ear.
The MRI report says “mild” or “moderate,” so the cord is dismissed.

But myelopathy is not diagnosed by one symptom, one reflex, or one word in a report. It is recognized by mapping the history, neurological examination, function, progression, and imaging together.

A routine MRI is a static snapshot taken lying down with the neck neutral. In selected patients, compression becomes more obvious with movement. Early cord dysfunction may also exist before imaging looks dramatic.

A vulnerable spinal cord should not be approached like routine neck stiffness. Forceful end-range positioning, aggressive manipulation, or repeatedly provoking neurological symptoms may be inappropriate until cord involvement has been assessed.

At Movability, I am always screening for this. I look at hand dexterity, gait, reflexes, balance, proprioception, strength, progression, and whether seemingly unrelated symptoms form one cord-level pattern.

I have caught this in patients who had no idea their spinal cord might be involved, including people whose symptoms had been dismissed or treated separately.

Sometimes the most important clinical decision is knowing when not to treat, and when to escalate for imaging, neurology, or a spine consultation.

My full breakdown is now in Movability Masterclass on Substack:

“Degenerative Cervical Myelopathy: The Hidden Progression From Silent Cord Compression to Permanent Injury.”

Link in bio.

Dr. Sina

08/05/2026

The migraine can become less painful and more confusing.

Someone may stop having one-sided, pounding attacks, yet start living with daily pressure, dizziness in busy places, nausea from screens, brain fog, neck discomfort, or a postdrome that outlasts the pain.

Then every symptom gets separated. The neck becomes one issue. The dizziness gets sent down an inner-ear pathway. The fatigue gets blamed on menopause or aging. The visual symptoms get called stress.

Sometimes those explanations are partly right. But they may still be pieces of one changing neurological pattern.

This is why, when a migraine patient comes to see me, I do not start with the last few weeks. I take the full timeline from childhood to today.

Motion sickness. Early headaches. Puberty. Menstrual patterns. Concussions. Pregnancy and postpartum changes. Sleep. Hormonal transitions. Medication use. Neck and jaw symptoms. Most importantly, when the pattern changed.

After more than a decade of working with migraine patients, treating thousands of them, and serving as a global complex case consultant, I have learned to recognize patterns that disappear when each stage of life is viewed in isolation.

Migraine is not one fixed headache. It can shift toward aura, pressure, dizziness, sensory overload, cognitive fatigue, autonomic symptoms, or chronic sensitivity.

Hormones can alter the threshold, but aging also changes sleep, pain regulation, sensory integration, circulation, medication tolerance, and recovery.

So the useful question is not only, “Do you still get migraines?”

It is, “What did they look like before, what do they look like now, and what changed in between?”

Sudden, persistent, progressive, or unfamiliar neurological changes still require medical assessment.

I mapped the physiology, red flags, phenotype patterns, and clinical sequencing in the Movability Masterclass article on Substack:

WHY MIGRAINE CHANGES WITH AGE

Read it through the link in my bio

When someone has several disconnected explanations but no clear map, this is exactly the type of complex migraine pattern I assess at Movability.

Sometimes the migraine did not disappear. It changed the way it speaks.

Dr. Sina

07/31/2026

“Your tests are normal” should never be translated into “your symptoms are not real.”

It should mean the tests did not detect the type or degree of dysfunction they were designed to detect.

That distinction is where many complex nerve cases get lost.

A standard workup is excellent at identifying fixed lesions, large-fiber damage, denervation, major structural compression, and other serious pathology. It is much less suited to answering questions like:

Does the nerve move normally through its entire pathway?

Does it become symptomatic only after repetition, load, or a specific position?

Are several smaller restrictions creating one cumulative problem?

Is the nerve’s recovery capacity being reduced by sleep, inflammation, metabolic health, nutrition, or previous injury?

This is why I do not stop at the body part that hurts or the test that came back normal. I look at the whole nerve pathway, the patient’s functional threshold, and what changes when the suspected interfaces are treated and immediately retested.

That test-retest process matters. It tells us whether the system is mechanically modifiable, whether the treatment target makes sense, and whether we should continue, change direction, investigate further, or refer out.

I am Dr. Sina Yeganeh, chiropractor and global complex case consultant. I work with patients whose symptoms have outgrown single-region explanations and whose conventional workups have not explained why function keeps failing.

My newest Movability Masterclass article on Substack is:

“Normal EMG. Normal MRI. Still Numb: Why Double Crush Syndrome Misses Multisite Nerve Dysfunction.”

Inside, I break down the physiology of nerve glide, blood flow, fascial interfaces, mechanotransduction, tissue remodelling, and why double crush is often too narrow. I also walk through a real clinical case and the pivot points that changed the outcome, without reducing the story to one tunnel or one treatment.

The full Root Cause Breakdown is linked in my bio.

At Movability, this is why our complex case assessments examine the whole pathway, retest function, and let the physiology determine the next step.

Dr. Sina

You were told your cerebral venous sinus is narrow.But were you ever told why?For many people, that is exactly where the...
07/27/2026

You were told your cerebral venous sinus is narrow.

But were you ever told why?

For many people, that is exactly where the investigation stops. They are shown an MRV, given a label, and left without an explanation for what narrowed it, whether it is driving their symptoms, or what should be investigated next.

One clinician says it is congenital and irrelevant. Another treats the narrowing as the entire disease.

But the same image can represent completely different physiology.

It may be a normal anatomical variant. It may only look narrow because of slow or turbulent flow. It may be collapsing under elevated pressure around the brain. It may contain a clot, scar, arachnoid granulation, or web. The resistance may even sit farther downstream in the jugular or central venous system.

Sometimes the sinus is the cause.
Sometimes it is the consequence.
Sometimes it starts as the consequence, then becomes an amplifier that helps sustain the pressure problem.
Sometimes it is incidental.

The scan may be showing where the system gave way, not where the problem began.

That distinction changes everything.

The question is not only, “How narrow is it?”

The better questions are:

What force narrowed it?
What created that force?
Is it functionally significant?
Is it fixed or potentially reversible?
What should change when the real driver is treated?

You are not unreasonable for wanting more than a label. A finding on a scan should open the investigation, not close it.

My new Movability Masterclass article is:

“You Were Told Your Cerebral Venous Sinus Is Narrow. The Investigation Should Not Stop There.”

Inside, I break down the root-cause physiology, how to distinguish transient pressure-dependent collapse from fixed obstruction, when narrowing may reverse, what imaging and pressure testing can tell us, where investigations commonly stop too early, and the seven-step framework I use for these cases.

The full Root Cause Breakdown is now live on Substack.

Movability Masterclass is for clinicians and curious minds who refuse surface explanations and want to understand what is driving complex symptoms.

Link in bio.

Dr. Sina

07/24/2026

Allow me to re-introduce myself.

I’m Dr. Sina Yeganeh, chiropractor, global complex case consultant, and cofounder of Movability.

My work is centred on complex chronic pain and nerve-related cases, especially the ones that do not fit neatly into a diagnosis, a body part, or a single discipline.

The cases where symptoms move, findings conflict, imaging tells only part of the story, and people have seen multiple providers without anyone stepping back to connect the full picture.

Complexity is not at the edge of my work. It is the centre of it. These are the cases I love.

I’ve had the privilege of working alongside doctors and clinicians around the world whose thinking has challenged, shaped, and sharpened my own.

Those experiences have influenced the way I approach difficult cases: question assumptions, recognize patterns across systems, and never reduce a person to one scan, one structure, or one symptom.

That philosophy is also why we built Movability the way we did.

Complex care should not be siloed. The difference is not simply having multiple disciplines under one roof. It is how our team communicates, collaborates, and brings different clinical perspectives into the same conversation.

A collection of services is not the same as integrated care. Our goal is to use the full potential of a team-based model to make better sense of complexity and create a more coordinated path forward.

For the next couple of hours only, I’m opening a Q&A specifically about NERVE PAIN.

Burning, tingling, numbness, electric or shooting pain, weakness, sciatica-like symptoms, or symptoms that feel difficult to explain.

A few boundaries before you ask:

This is for general education only. I cannot diagnose you, assess your individual case, or provide personal medical advice through Instagram.

Please keep questions focused on nerve pain, and do not post private health information or imaging.

For clinic, booking, pricing, insurance, scheduling, or administrative questions, please visit our website or contact the clinic. Our team will be happy to help.

I’ll answer as many questions as I can before the Q&A closes.

Ask below. Let’s connect some dots.

07/22/2026

The psychiatric symptom may be the last event in the chain.

That is what medicine keeps missing about PMDD.

When bloating, reflux, broken sleep, migraine, flushing, palpitations, and sensory overload rise before rage or despair, I do not see a collection of unrelated diagnoses.

I see a sequence.

And in complex physiology, sequence is evidence.

Histamine matters because it is not only an allergy mediator. In the brain, it is a neurotransmitter that helps regulate wakefulness, arousal, attention, and sensory gain. In the body, mast-cell histamine affects the gut, blood vessels, airways, skin, and nerves.

Those histamine systems are not identical, but they are part of a body that is constantly communicating.

This is why women reporting major PMDD changes after H1 and H2 blockers deserve more than dismissal.

Not because they have proven a treatment.

Because their responses may have exposed a convergence point.

If altering histamine signalling shifts mood, sleep, migraine, digestion, flushing, and autonomic symptoms together, the most interesting question is not:

“Which pill worked?”

It is:

“What network did we just expose?”

This is the work I do in complex cases. I reconstruct physiology in motion. I look for the earliest shift, the biological timing, the systems that move together, and the difference between a trigger, an amplifier, and a maintainer.

The hormone transition may be the trigger.

The gut-immune load may be the amplifier.

Sleep loss and migraine may become maintainers.

Histamine may be one of the bridges.

This is not a story about “high histamine” or the gut simply causing PMDD.

It is a more sophisticated possibility:

A hormonally sensitive nervous system may be pushed across a threshold by immune, gut, vascular, sensory, and autonomic load.

The mood symptom may be the final expression of a whole-body state.

That is a very different way to read the case.

I unpack the full model, evidence, sequencing logic, and clinical case in Movability Masterclass on Substack.

Read through the link in my bio:

“PMDD and Histamine: How the Gut-Immune-Hormone Axis May Shape Cyclical Mood Symptoms.”

Dr. Sina

A complex case does not mean a difficult person. Often, it means the explanation so far has been too small.Movability wa...
07/20/2026

A complex case does not mean a difficult person. Often, it means the explanation so far has been too small.

Movability was built from moments that forced us to look again: a diagnosis that did not explain the whole story, a plan that stopped working, and a person who had told their story repeatedly but never felt fully understood.

Those moments shaped our standards.

We learned that good care is not about becoming attached to the first answer. It is about staying curious enough to earn a better one. That means listening before assuming, questioning a label when it no longer fits, and using a systems-based approach to understand the pattern as a whole, not only where it becomes visible.

Not complexity for its own sake. The goal is clarity.

A symptom may be felt in one place without being explained by that place alone. The place that hurts still matters, but it may not tell the whole story.

Our role is not to force every case into the same explanation, or promise one hidden cause behind every symptom. It is to ask better questions, make the reasoning clear, involve you in the process, and change course when new information changes the picture.

Credibility does not come from certainty alone. It comes from reasoning that can be explained, questioned, and revised as the evidence changes.

When you have lived with a problem that has never been fully explained, clarity is not just reassuring. It gives you somewhere to begin. It restores direction.

Perhaps that is why people from around the world keep finding their way to Movability.

Beneath many of those stories is the same question:

What happens when the usual answers are no longer enough?

That question can begin a different kind of search. Not for another quick fix, but for a clearer understanding of the pattern, how its parts relate, and what can meaningfully change.

Movability is built around attention, collaboration, and the belief that complex problems rarely fit a standard script.

The difficult moments did not make us cynical. They made us more deliberate about the kind of clinic we wanted to become.

We did not build Movability simply to treat more people.

We built it to see people more completely.

07/17/2026

Her neck looked like the problem. I did not touch it.

She came in with left-sided neck and shoulder tightness. The tension climbed into the base of her skull and contributed to head and facial pain.

When I tested her neck, bending to the right was restricted because the left side would not release.

But the most important finding was not in her neck.

After a stressful couple of weeks, her left TMJ and chewing muscles were tighter than the right, matching her neck tension and headache.

I treated only her left jaw with soft-tissue work, neural techniques, and joint mobilization. I avoided her neck and shoulder to create a clinical test.

Then we retested.

Her right side-bending improved dramatically. The left-sided guarding dropped, and her neck moved more freely without being touched.

How can TMJ treatment change neck range of motion?

The jaw and upper neck are not isolated systems. Sensory information from the teeth, TMJ, and chewing muscles enters the trigeminal system, where it interacts with input from the upper cervical nerves.

When the jaw feels painful or restricted, the nervous system may increase jaw and neck co-contraction. A “tight” neck muscle may partly be a protective response to information coming from the jaw.

As a global complex case consultant, I’ve been fortunate to collaborate with dentists, airway specialists, and TMJ specialists worldwide. Those cases reinforced the need to view the jaw, bite, airway, head, and neck as one connected system.

Change the input, and the output can change.

This does not mean every neck problem comes from the TMJ or that every patient needs bite correction. It means the jaw was a meaningful contributor to this patient’s pattern.

The painful area and the primary driver are not always the same place.

Save this for the next time neck tension, headaches, jaw tightness, clenching, dental work, or bite changes appear together. Share it with someone who keeps treating the neck without checking the jaw.

Read the full Root Cause Breakdown on my Substack, Movability Masterclass: “When the Jaw Drives the Neck: The Neuroanatomy of TMJ Dysfunction, Dental Work, Bite Changes, and Headache”

Link in bio

Dr. Sina

They didn’t become a different person. Their body had been living in “agita.”Their family says, “You’re not yourself.”A ...
07/15/2026

They didn’t become a different person. Their body had been living in “agita.”

Their family says, “You’re not yourself.”

A calm person becomes wired, anxious, irritable and short-fused. They clench their jaw, wake with headaches, battle TMJ pain, notice tinnitus and may see blood pressure rise when pain and fight-or-flight take over. Gastritis, reflux, gas and upper-abdominal pressure can feel like palpitations or chest discomfort. Every sensation triggers another alarm until they stop trusting their body.

Too often, they hear, “It’s just stress.”

Then we test, and there it is: H. pylori.

Sometimes it appears on a GI-MAP qPCR stool panel. But the result is only one piece. We listen to the timeline and what the patient has been trying to tell us.

H. pylori lives in the stomach, but the fallout can be felt throughout the gut. Inflammation, altered digestion, poor sleep, gut-brain signalling and a nervous system stuck in survival mode can ripple far beyond it.

Eradication often requires antibiotics and acid suppression. That may eliminate the infection while causing nausea, bloating, altered stools, food intolerance and microbiome disruption.

That is why we never confuse eradication with recovery.

A negative test tells us the infection is gone. It does not tell us whether the stomach lining has healed, digestion is normal and the gut ecosystem has recovered.

This is where systems-based care matters.

We confirm eradication. We assess the stomach and whole GI tract. We review medications, food tolerance, bowel function, nutrition, sleep, jaw tension, stress physiology and microbiome findings. Then we rebuild brick by brick, with a plan designed for the person in front of us, not a random pile of supplements.

Then they say, “I feel like myself again.”

Sometimes the personality never changed. Their body sounded the alarm for so long that survival mode became normal.

If someone you know started acting like Tony Soprano when their gut fell apart, send them this. The “agita” may not be who they are. It may be what H. pylori, gastritis and a nervous system stuck on high alert have done to them.

Chest pain requires prompt medical evaluation.

Dr. Sina

Address

2 Hunters Point Drive
Richmond Hill, ON
L4C9Y4

Opening Hours

Monday 10am - 7pm
Tuesday 10am - 7pm
Wednesday 10am - 7pm
Thursday 10am - 7pm
Friday 10am - 7pm
Saturday 10am - 4pm
Sunday 10am - 4pm

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+19057634000

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