Anna Risius Physio

18/06/2026

🚴‍♀️🐎 600km. 3 days. 2 incredible causes. 💙

This summer, a group of well-known faces from across the eventing community will be swapping their usual roles for life on two wheels as they take on an epic 600km cycle challenge from Scone Palace International Horse Trials to Defender Burghley Horse Trials in just three days.

Taking on hundreds of miles, challenging terrain and long hours in the saddle, they'll be raising vital funds for both the British Eventing Support Trust and Riders Minds.

Every pedal stroke will help support people within the equestrian community through physical recovery, emotional wellbeing and mental health challenges. Together, these two charities provide invaluable support when it's needed most.

This is no small undertaking. Covering 600km in just three days will require determination, resilience and a huge team effort, and we couldn't be more grateful to everyone taking part.

Over the coming weeks, we'll be introducing the cyclists, sharing their preparations and bringing you updates as they make their way from Scotland to one of eventing's most iconic destinations.

💙 If you'd like to support the team and help raise funds for these two fantastic charities, you can donate here: https://www.givewheel.com/fundraising/17381/scone-to-burghley/

For now, we'd like to say a huge thank you to everyone involved. We can't wait to follow the journey, cheer them on every mile of the way and see them roll into Burghley after what promises to be an unforgettable challenge. 🚴‍♂️🙌

Hi Ho Silver
FAIRFAX & FAVOR
1st Class Images
Piggy March
Nicola Wilson Equestrian

10/06/2026

Hello!
An introduction for new visitors here;

I'm Anna, a Chartered Physiotherapist with over 20 years experience in optimising the musculoskeletal health of people and animals.
My training and experience allows me to apply physiotherapy principles to any species, but my work generally is with people, horses and dogs. My passion is equestrian sport and I have been privileged to work with human and equine athletes at the highest level, but also get huge rewards from helping anyone out of pain and improving the quality of life of our canine companions.
My increasingly sporty family also keep me on my toes regarding health and fitness!
People and dogs come here to my base just outside Somerton, Somerset and I go out to visit horses within a 40 mile radius.
I offer appointments Monday to Friday 9am-4pm with flexibility for days/times subject to availability and planning ahead.

For apointments and queries feel free to get in touch!
07841 877660 [email protected]

17/05/2026

Just published 🔥

𝗧𝗵𝗲 𝗲𝗳𝗳𝗶𝗰𝗮𝗰𝘆 𝗼𝗳 𝘀𝘁𝗿𝗲𝗻𝗴𝘁𝗵 𝗲𝘅𝗲𝗿𝗰𝗶𝘀𝗲 𝗱𝗼𝘀𝗮𝗴𝗲 𝗼𝗻 𝗽𝗮𝗶𝗻 𝗮𝗻𝗱 𝗱𝗶𝘀𝗮𝗯𝗶𝗹𝗶𝘁𝘆 𝗶𝗻 𝗽𝗲𝗼𝗽𝗹𝗲 𝘄𝗶𝘁𝗵 𝗹𝗼𝘄 𝗯𝗮𝗰𝗸 𝗽𝗮𝗶𝗻: 𝗮 𝘀𝘆𝘀𝘁𝗲𝗺𝗮𝘁𝗶𝗰 𝗿𝗲𝘃𝗶𝗲𝘄 𝗼𝗳 𝗿𝗮𝗻𝗱𝗼𝗺𝗶𝘇𝗲𝗱 𝗰𝗼𝗻𝘁𝗿𝗼𝗹𝗹𝗲𝗱 𝘁𝗿𝗶𝗮𝗹𝘀

🏋️‍♂️ Although strength exercises are recommended for for back pain (LBP) (https://pmc.ncbi.nlm.nih.gov/articles/PMC8477273/, https://pubmed.ncbi.nlm.nih.gov/40624581/), more information is needed on how to prescribe them. To date, no systematic review (SR) has investigated the impact of strength exercise parameters (e.g., intensity, frequency, sets, repetitions, exercise type, and treatment duration) on outcomes for LBP.

📘 A brand-new SR of randomized controlled trials (RCTs) by de Oliveira et al. (https://link.springer.com/article/10.1007/s00586-026-09901-5) aimed to investigate the efficacy of strength exercises dosages on pain intensity and disability in people with LBP. Comparisons of interest were placebo, sham, waiting list, and no intervention to clarify exercise efficacy.

📋 Although the review question was broad, the included evidence ultimately applied only to people with chronic non-specific low back pain; no eligible trials involved acute, subacute, radicular, or specific spinal conditions. Eight randomized controlled trials were included in the qualitative synthesis, and five contributed to the meta-analysis.

Main clinical finding

📊 Strength exercise showed statistically significant short-term improvements in both pain and disability compared with minimal intervention. The pooled effects were:

⬇️ Pain intensity: MD −15.89 points on a 0–100 scale, 95% CI −27.66 to −4.13
⬇️ Disability: MD −9.30 points on a 0–100 scale, 95% CI −13.80 to −4.81

✅ These effects suggest a small to moderate short-term benefit, with the pain effect approaching a clinically meaningful range depending on the threshold used. However, the certainty of evidence was low to very low, so the estimates are uncertain.

𝗗𝗼𝘀𝗮𝗴𝗲-𝘀𝗽𝗲𝗰𝗶𝗳𝗶𝗰 𝗲𝗳𝗳𝗲𝗰𝘁𝘀

🏋️‍♂️ The most consistent effects were seen for high-intensity, low-frequency, lumbar extensor-focused strength training.

▶️ 𝗛𝗶𝗴𝗵 𝗶𝗻𝘁𝗲𝗻𝘀𝗶𝘁𝘆 >60% 1𝗥𝗠

High-intensity strength exercise produced statistically significant effects:
⬇️ Pain: MD −19.3, 95% CI −27.0 to −11.6
⬇️ Disability: MD −14.2, 95% CI −15.7 to −12.8

✅ This was one of the strongest signals in the review and suggests that progressive loading above 60% 1RM may be more effective than lower-intensity strengthening for chronic non-specific low back pain.

𝗟𝗼𝘄 𝘄𝗲𝗲𝗸𝗹𝘆 𝗳𝗿𝗲𝗾𝘂𝗲𝗻𝗰𝘆: 𝗼𝗻𝗰𝗲 𝗽𝗲𝗿 𝘄𝗲𝗲𝗸

Once-weekly strength training was associated with significant improvements:

⬇️ Pain: MD −19.3, 95% CI −27.0 to −11.6
⬇️ Disability: MD −11.8, 95% CI −16.2 to −7.5

✅ Training more than once per week did not show the same consistent effect. Clinically, this may indicate that higher-load lumbar strengthening does not necessarily require high weekly frequency, possibly because recovery and tolerability are important in this population.

𝗦𝗵𝗼𝗿𝘁 𝘁𝗿𝗲𝗮𝘁𝗺𝗲𝗻𝘁 𝗱𝘂𝗿𝗮𝘁𝗶𝗼𝗻: ≤3 𝗺𝗼𝗻𝘁𝗵𝘀

Shorter programs showed significant effects:

⬇️ Pain: MD −19.9, 95% CI −26.0 to −13.8
⬇️ Disability: MD −13.4, 95% CI −15.2 to −11.6

✅ Longer interventions did not show statistically significant pooled effects, but this should not be interpreted as evidence that longer treatment is ineffective. The number of trials was small, and the evidence was imprecise.

𝗟𝘂𝗺𝗯𝗮𝗿 𝗲𝘅𝘁𝗲𝗻𝘀𝗶𝗼𝗻- 𝗲𝘅𝗲𝗿𝗰𝗶𝘀𝗲𝘀

Lumbar extension exercises showed clearer benefits than general strength training:

⬇️ Pain: MD −19.6, 95% CI −25.4 to −13.7
⬇️ Disability: MD −12.5, 95% CI −14.9 to −10.1

✅ General strength training alone showed smaller and non-significant effects in the subgroup analyses

⬇️ Pain: MD −8.0, 95% CI −27.5 to 11.6
⬇️ Disability: MD −3.2, 95% CI −7.8 to 1.4

✅ This does not mean general strength training is ineffective, but the available evidence more strongly supports targeted lumbar extensor strengthening.

𝗟𝗼𝗻𝗴-𝘁𝗲𝗿𝗺 𝗲𝗳𝗳𝗲𝗰𝘁𝘀

Long-term effects were not statistically significant:

⬇️ Pain: MD −9.27, 95% CI −22.83 to 4.27
⬇️ Disability: MD −2.85, 95% CI −7.13 to 1.43
•
✅ Therefore, the evidence mainly supports short-term improvement, while sustained effects remain unclear.

𝗣𝗿𝗮𝗰𝘁𝗶𝗰𝗮𝗹 𝗶𝗻𝘁𝗲𝗿𝗽𝗿𝗲𝘁𝗮𝘁𝗶𝗼𝗻 💡

For physiotherapists treating patients with chronic non-specific low back pain, the review supports the use of progressive strength training, especially programs that target the lumbar extensors and reach higher relative intensities when clinically tolerated. A clinically reasonable interpretation is that once-weekly, high-intensity lumbar extensor strengthening over a period of up to 12 weeks may reduce pain by roughly 19–20 points on a 0–100 scale and disability by roughly 12–14 points on a 0–100 scale. These are meaningful short-term changes, but they should be applied with clinical reasoning because the evidence certainty is low.

17/05/2026

𝗡𝗲𝘄 𝗿𝗲𝘀𝗲𝗮𝗿𝗰𝗵 𝗿𝗲𝘃𝗲𝗮𝗹𝘀 𝗵𝗼𝘄 𝗔𝗰𝗵𝗶𝗹𝗹𝗲𝘀 𝘁𝗲𝗻𝗱𝗶𝗻𝗼𝗽𝗮𝘁𝗵𝘆 𝗿𝗲𝘄𝗶𝗿𝗲𝘀 𝘆𝗼𝘂𝗿 𝗯𝗿𝗮𝗶𝗻'𝘀 𝗺𝘂𝘀𝗰𝗹𝗲 𝗰𝗼𝗻𝘁𝗿𝗼𝗹 𝘀𝘁𝗿𝗮𝘁𝗲𝗴𝘆!

A recent 2025 study in 𝗧𝗵𝗲 𝗝𝗼𝘂𝗿𝗻𝗮𝗹 𝗼𝗳 𝗣𝗵𝘆𝘀𝗶𝗼𝗹𝗼𝗴𝘆 investigated how Non-Insertional Achilles Tendinopathy (NIAT) impacts the central nervous system's control of the triceps surae (calf) muscles.

While NIAT physically thickens and reduces the stiffness of the Achilles tendon, the research shows it also dramatically changes how the brain activates the calf muscles in a load-dependent manner.

🔍 𝗞𝗲𝘆 𝗙𝗶𝗻𝗱𝗶𝗻𝗴𝘀

𝗧𝗵𝗲 𝗟𝗮𝘁𝗲𝗿𝗮𝗹 𝗚𝗮𝘀𝘁𝗿𝗼𝗰𝗻𝗲𝗺𝗶𝘂𝘀 (𝗟𝗚) 𝗴𝗼𝗲𝘀 𝗶𝗻𝘁𝗼 𝗼𝘃𝗲𝗿𝗱𝗿𝗶𝘃𝗲

At high force levels, the LG muscle exhibits a significantly increased motor unit discharge rate and a delayed de-recruitment threshold.

This means the muscle is commanded to work much harder and stays active longer compared to healthy individuals.

𝗥𝗲𝗱𝘂𝗰𝗲𝗱 𝗺𝘂𝘀𝗰𝗹𝗲 𝘁𝗲𝗮𝗺𝘄𝗼𝗿𝗸 𝗮𝘁 𝗹𝗼𝘄 𝗳𝗼𝗿𝗰𝗲𝘀

During low-force tasks, there is decreased functional connectivity and coordination between the medial gastrocnemius (MG) and the LG.

This indicates a highly uneven distribution of neural drive to the calf muscles.

𝗔𝗹𝘁𝗲𝗿𝗲𝗱 𝘁𝗼𝗿𝗾𝘂𝗲 𝗰𝗼𝗻𝘁𝗿𝗶𝗯𝘂𝘁𝗶𝗼𝗻

The LG's contribution to your overall ankle push-off force is significantly reduced during low-force tasks, but it shifts to become highly involved during high-force tasks.

📃 𝗪𝗵𝘆 𝗱𝗼𝗲𝘀 𝘁𝗵𝗶𝘀 𝗺𝗮𝘁𝘁𝗲𝗿?

These muscle-specific compensations are likely a subconscious strategy to manage pain and adapt to the injured tendon's altered mechanical properties.

However, because the calf muscles share the Achilles tendon, this uneven muscle activation can cause uneven force transmission and heterogeneous loading across the tendon itself.

By placing disproportionate stress on specific parts of the tendon, these neural adaptations might actually perpetuate or worsen the tendinopathy.

📌 𝗖𝗹𝗶𝗻𝗶𝗰𝗮𝗹 𝗜𝗺𝗽𝗹𝗶𝗰𝗮𝘁𝗶𝗼𝗻

Understanding that 𝗧𝗵𝗲 𝗹𝗮𝘁𝗲𝗿𝗮𝗹 𝗴𝗮𝘀𝘁𝗿𝗼𝗰𝗻𝗲𝗺𝗶𝘂𝘀 𝗽𝗹𝗮𝘆𝘀 𝗮 𝗰𝗲𝗻𝘁𝗿𝗮𝗹 𝗿𝗼𝗹𝗲 𝗶𝗻 𝘁𝗵𝗲 𝗽𝗮𝘁𝗵𝗼𝗽𝘆𝘀𝗶𝗼𝗹𝗼𝗴𝘆 𝗼𝗳 𝗡𝗜𝗔𝗧 highlights the need to consider neuromuscular control—not just tendon structure—when designing precise rehabilitation strategies for chronic Achilles pain.

💬 𝗣𝗵𝘆𝘀𝗶𝗰𝗮𝗹 𝗧𝗵𝗲𝗿𝗮𝗽𝘆 𝗘-𝗟𝗲𝗮𝗿𝗻𝗶𝗻𝗴 𝗖𝗼𝗺𝗺𝗲𝗻𝘁

👉 This study adds to the growing evidence that Achilles tendinopathy is not only a tendon pathology, but also a neuromuscular control disorder.

👉 The findings suggest that the nervous system may redistribute load between calf muscles in an attempt to protect the painful tendon, yet these compensations could unintentionally maintain abnormal tendon loading over time.

👉 Understanding these muscle-specific neural adaptations may help clinicians move beyond purely structural approaches and toward more targeted rehabilitation strategies focused on restoring balanced calf muscle function and load distribution.

■■ 𝗖𝗹𝗶𝗻𝗶𝗰𝗮𝗹 𝗜𝗺𝗽𝗹𝗶𝗰𝗮𝘁𝗶𝗼𝗻𝘀 𝗶𝗻 𝗿𝗲𝗵𝗮𝗯𝗶𝗹𝗶𝘁𝗮𝘁𝗶𝗼𝗻

• Rehabilitation may need to address neuromuscular control deficits in addition to tendon loading capacity.

• The lateral gastrocnemius may represent an important treatment target during both assessment and exercise prescription.

• Load-specific rehabilitation strategies could be important, as muscle behavior appears to differ between low-force and high-force tasks.

• Improving coordination between the medial and lateral gastrocnemius may help optimize force distribution across the Achilles tendon.

• Persistent symptoms in NIAT may partly reflect altered central nervous system motor strategies rather than tendon structure alone.

25/04/2026

𝗧𝗵𝗲 𝗚𝘂𝘁-𝗗𝗶𝘀𝗸 𝗔𝘅𝗶𝘀: 𝗛𝗼𝘄 𝗬𝗼𝘂𝗿 𝗠𝗶𝗰𝗿𝗼𝗯𝗶𝗼𝗺𝗲 𝗠𝗶𝗴𝗵𝘁 𝗕𝗲 𝗗𝗿𝗶𝘃𝗶𝗻𝗴 𝗖𝗵𝗿𝗼𝗻𝗶𝗰 𝗟𝗼𝘄 𝗕𝗮𝗰𝗸 𝗣𝗮𝗶𝗻

⬛ Chronic low back pain (LBP) is a massive global health issue, affecting an estimated 500 million people and acting as the leading cause of disability worldwide.
⬛ Traditionally, we have viewed back pain as a purely structural or mechanical problem—a slipped disk, a pinched nerve, or joint wear and tear.
⬛ However, many patients suffer from debilitating chronic pain without any obvious structural damage that would require surgery.

⬛ A groundbreaking 2026 pilot study by Sima et al., published in JOR Spine, sheds new light on a hidden culprit: the gut microbiome.
⬛ By exploring the emerging concept of the "gut-disk axis," researchers are uncovering how an imbalance in our gut bacteria might be fueling systemic inflammation and driving back pain, even in structurally "healthy" spines.

🟦 𝗧𝗵𝗲 𝗦𝘁𝘂𝗱𝘆: 𝗟𝗼𝗼𝗸𝗶𝗻𝗴 𝗕𝗲𝘆𝗼𝗻𝗱 𝘁𝗵𝗲 𝗦𝗽𝗶𝗻𝗲

⬛ Prior research has hinted at a connection between gut dysbiosis (microbial imbalance) and inflammatory conditions like rheumatoid arthritis and osteoarthritis.
⬛ To specifically test this link in back pain, the researchers conducted a case-control study matching 28 patients suffering from chronic LBP (lasting more than 3 months) with 28 healthy controls.

⬛ Crucially, the LBP patients in this study did not have advanced disk degeneration or conditions requiring surgical intervention.
⬛ The groups were rigorously matched for age, s*x, and Body Mass Index (BMI) to isolate the microbiome's specific role.
⬛ Researchers analyzed the participants' stool samples using advanced 16S rRNA sequencing to profile their gut bacteria.

🟦 𝗞𝗲𝘆 𝗙𝗶𝗻𝗱𝗶𝗻𝗴𝘀: 𝗔 𝗗𝗶𝘀𝗿𝘂𝗽𝘁𝗲𝗱 𝗠𝗶𝗰𝗿𝗼𝗯𝗶𝗮𝗹 𝗘𝗰𝗼𝘀𝘆𝘀𝘁𝗲𝗺

⬛ Reduced Alpha Diversity: LBP patients exhibited a significantly lower "alpha diversity".
⬛ This metric refers to the richness and evenness of bacterial species in the gut; a lower score indicates a less complex microbial environment, which is a classic hallmark of dysbiosis.

⬛ Distinct Community Structures (Beta Diversity): The overall makeup of the bacterial communities between the healthy controls and LBP patients clustered into distinctly different groups, indicating a fundamental shift in the microbiome associated with chronic pain.

⬛ Depletion of "Good" Bacteria: Beneficial microbes were significantly reduced in LBP patients.
⬛ At the phylum level, Bacteroidota was depleted.
⬛ At the genus level, Parabacteroides saw a significant decrease.
⬛ Both of these bacteria are vital producers of short-chain fatty acids (SCFAs), such as butyrate, which are essential for maintaining the gut barrier and suppressing inflammation.

⬛ Overgrowth of "Bad" Bacteria (Pathobionts): LBP patients had significantly elevated levels of Proteobacteria and Desulfobacterota.
⬛ Proteobacteria overgrowth is known to trigger severe inflammation and disrupt intestinal tight junctions.
⬛ Desulfobacterota produces hydrogen sulfide, which at high levels is toxic to gut cells and contributes to a "leaky gut".
⬛ Additionally, Prevotella—a bacteria linked to low-grade systemic inflammation and activated immune responses—was significantly elevated in the chronic pain group.
⬛ (Note: The study also found elevated levels of Faecalibacterium, which was unexpected as it is generally considered anti-inflammatory, suggesting the need for further strain-specific research).

🟦 𝗧𝗵𝗲 𝗠𝗲𝗰𝗵𝗮𝗻𝗶𝘀𝗺: 𝗨𝗻𝗱𝗲𝗿𝘀𝘁𝗮𝗻𝗱𝗶𝗻𝗴 𝘁𝗵𝗲 "𝗚𝘂𝘁-𝗗𝗶𝘀𝗸 𝗔𝘅𝗶𝘀"

🔗 ⬛ How exactly does an unhealthy gut cause a sore back? The researchers propose the "gut-disk axis" framework.

⬛ When the gut microbiome loses its diversity and beneficial SCFA-producing bacteria decline, the structural integrity of the gut lining weakens.
⬛ This loosening of the epithelial "tight junctions" leads to increased intestinal permeability, commonly known as "leaky gut".

⬛ Once the gut barrier is compromised, endotoxins (like lipopolysaccharides) and bacteria can escape the intestines and translocate into the systemic bloodstream.
⬛ This systemic endotoxemia triggers the immune system to release massive amounts of pro-inflammatory cytokines, such as IL-6, TNF-α, and IL-17.
⬛ These inflammatory molecules—and sometimes the bacteria themselves—travel through the bloodstream to the intervertebral disks in the spine.
⬛ There, they drive localized inflammation, neural infiltration, nociceptive (pain) sensitization, and ultimately, disk degeneration.

🟦 𝗪𝗵𝗮𝘁 𝗧𝗵𝗶𝘀 𝗠𝗲𝗮𝗻𝘀 𝗳𝗼𝗿 𝘁𝗵𝗲 𝗙𝘂𝘁𝘂𝗿𝗲 𝗼𝗳 𝗕𝗮𝗰𝗸 𝗣𝗮𝗶𝗻 𝗧𝗿𝗲𝗮𝘁𝗺𝗲𝗻𝘁

📌 ⬛ These findings represent a massive paradigm shift.
⬛ If chronic low back pain in non-surgical patients is partially driven by a dysfunctional gut, then traditional treatments like painkillers or spinal procedures may only be masking the symptoms rather than addressing the root cause.

⬛ By identifying specific microbial signatures associated with LBP, this research opens the door to microbiome-targeted therapies.

⬛ In the future, back pain management could heavily feature interventions aimed at restoring gut balance, such as:
⬛ Targeted probiotics and prebiotics
⬛ Dietary modifications aimed at boosting SCFA-producing bacteria
⬛ F***l microbiota transplantation (FMT)
⬛ Therapies like physiotherapy and cognitive behavioral therapy, which emerging evidence suggests may partly relieve pain by positively altering the gut-brain-disk axis

⬛ While this cross-sectional study cannot definitively prove causation, it strongly supports the idea that your digestive health and your spinal health are intimately connected.
⬛ Taking care of your gut might just be the secret to taking care of your back.

18/03/2026

14/03/2026

I was talking about this research with a patient the other day!

I feel it’s not how you lift, it’s how much and how frequently you lift, and making sure your body is fit for whatever tasks you are doing 💪👊👍

𝗧𝗼 𝗙𝗹𝗲𝘅 𝗼𝗿 𝗡𝗼𝘁 𝘁𝗼 𝗙𝗹𝗲𝘅? 𝗗𝗲𝗯𝘂𝗻𝗸𝗶𝗻𝗴 𝘁𝗵𝗲 "𝗦𝘁𝗿𝗮𝗶𝗴𝗵𝘁 𝗕𝗮𝗰𝗸" 𝗟𝗶𝗳𝘁𝗶𝗻𝗴 𝗠𝘆𝘁𝗵

◼️ Low back pain (LBP) is the leading cause of disability worldwide, and lifting objects is commonly blamed as a primary risk factor.
◼️ For decades, workplace safety guidelines and healthcare professionals have preached a universal rule: avoid bending your spine and lift with a "straight back" to prevent injury.
◼️ But what if this deeply ingrained public health dogma isn't actually supported by science?
◼️ A fascinating systematic review and meta-analysis by Saraceni et al. (2020) set out to investigate the core idea behind this rule.
◼️ The researchers asked two fundamental questions: Is lifting with a flexed lumbar spine actually a risk factor for developing or worsening LBP? And do people with LBP actually bend their spines more during lifting than people without pain?
◼️ Here is a deep dive into the idea behind the review, what the researchers found, and why it might change how we think about lifting.

🧠 𝗧𝗵𝗲 𝗢𝗿𝗶𝗴𝗶𝗻 𝗼𝗳 𝘁𝗵𝗲 "𝗦𝘁𝗿𝗮𝗶𝗴𝗵𝘁 𝗕𝗮𝗰𝗸" 𝗗𝗼𝗴𝗺𝗮

◼️ The advice to maintain a straight back or neutral spine while lifting didn't come from nowhere.
◼️ It was largely based on early cadaveric studies, which demonstrated that a dead spine is more susceptible to structural failure when it is repeatedly flexed and compressed at the same time.
◼️ However, dead tissues do not accurately represent living, breathing human bodies.
◼️ The researchers noted that we do not fully understand how applicable these cadaver studies are to real-life, in vivo (living) lifting situations.
◼️ In fact, recent biomechanical studies on living humans show that spinal loads (disc pressure, compression, and shear strain) are actually very similar whether you lift with a flexed spine or a straight spine.

📊 𝗪𝗵𝗮𝘁 𝘁𝗵𝗲 𝗗𝗮𝘁𝗮 𝗔𝗰𝘁𝘂𝗮𝗹𝗹𝘆 𝗦𝗵𝗼𝘄𝘀

◼️ To test the idea that spinal flexion is dangerous, the researchers analyzed 12 independent studies involving nearly 700 participants.
◼️ They compared the lumbar spine kinematics (movement) of people with LBP to those without it during natural, unconstrained lifting tasks.
◼️ The findings were highly consistent and directly challenge conventional wisdom.
🔹 No increased risk:
◼️ There is no credible longitudinal or cross-sectional evidence showing that greater lumbar spine flexion during lifting is a risk factor for the onset or persistence of LBP.
🔹 No significant difference:
◼️ Most studies found absolutely no difference in peak lumbar spine flexion between people who have LBP and people who are pain-free.
🔹 Less flexion, not more:
◼️ When differences were observed, the data actually trended in the opposite direction.
◼️ Several cross-sectional studies revealed that people with LBP often lifted with less lumbar flexion than healthy controls—likely as a protective adaptation to pain or because they were previously advised to keep their backs straight.
◼️ In short, out of 43 different lifting comparisons analyzed in the review, only two suggested that people with LBP used more spinal flexion, while the vast majority showed no difference or less flexion.

⚠️ 𝗪𝗵𝘆 𝗧𝗵𝗶𝘀 𝗠𝗮𝘁𝘁𝗲𝗿𝘀: 𝗧𝗵𝗲 𝗗𝗮𝗻𝗴𝗲𝗿 𝗼𝗳 𝗙𝗲𝗮𝗿

◼️ The idea behind this review extends beyond just body mechanics; it touches on psychology.
◼️ LBP is strongly influenced by biopsychosocial factors, including negative beliefs and a fear of movement.
◼️ When authorities constantly warn people that bending their back is dangerous, it can create a pervasive fear of normal human movement.
◼️ Because there is no in vivo evidence to support the warning that lumbar flexion causes LBP, the researchers argue that continuing to prescribe the "straight back" advice is unjustified and potentially harmful if it makes people unnecessarily afraid to move their bodies.

⚖️ 𝗡𝘂𝗮𝗻𝗰𝗲 𝗮𝗻𝗱 𝗟𝗶𝗺𝗶𝘁𝗮𝘁𝗶𝗼𝗻𝘀

◼️ While the idea that we don't need to fear spinal flexion is liberating, the review does come with a few important caveats.
🔹 Low-Quality Evidence:
◼️ The researchers graded the overall quality of the available evidence as "low," largely because biomechanical studies often use small sample sizes and different motion-capture methods.
🔹 Weight Limits:
◼️ The studies included in the review only tested participants lifting objects weighing up to 12 kg (about 26.5 lbs).
◼️ Therefore, these findings confidently apply to everyday lifting, but we cannot safely extrapolate them to heavy occupational lifting or maximum-effort weightlifting.
🔹 Laboratory Settings:
◼️ All the analyzed studies took place in labs, meaning we need more field-based research to see how these mechanics play out during repeated manual labor in actual workplaces.

✅ 𝗧𝗵𝗲 𝗧𝗮𝗸𝗲𝗮𝘄𝗮𝘆

◼️ The central idea of this comprehensive review is a paradigm shift: there is no credible evidence that bending your lower back when you lift causes low back pain.
◼️ While we still need more research on heavy occupational lifting, the current science suggests that the human spine is robust and capable of bending safely.
◼️ Instead of fearing flexion and forcing a rigid "straight back," we may need to rethink our approach to manual handling and focus on building resilience rather than restricting natural movement.

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⚠️Disclaimer: Sharing a study or a part of it is NOT an endorsement. Please read the original article and evaluate critically.⚠️

Link to Article 👇

31/12/2025

Happy New Year! 🥳

We are moving from the year of the snake 🐍 to the year of the horse 🐴 - I am hopeful!

Looking forward to working with you all in 2026

🐾🐎🏃‍♀️‍➡️🙋‍♀️

23/12/2025

Wishing everyone a very merry Christmas!🎄🎅🎉