Dr.Abbas Sader

Dr.Abbas Sader This page was created by AS sader (Abbas sader), providing some clinical informations
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20/03/2025

Carbon monoxide poisoning from sh**ha smoking represents a significant yet underrecognized health hazard. The charcoal used to heat the to***co produces dangerous levels of this odorless, colorless gas, which can lead to a range of symptoms from headaches and dizziness to unconsciousness and, in severe cases, death. The misconception that water filtration makes ho**ah smoking safe has contributed to risky behaviors, particularly among young adults who represent the demographic most likely to engage in this activity.

The increasing prevalence of ho**ah-related carbon monoxide poisoning cases in recent years highlights the urgent need for greater public awareness about this risk. Healthcare providers, public health officials, and educators should work to disseminate accurate information about the dangers of carbon monoxide exposure from sh**ha smoking. Additionally, individuals who choose to smoke ho**ah should take precautions to minimize their risk by ensuring proper ventilation and being alert to symptoms of carbon monoxide poisoning.

As research continues to document the connection between sh**ha smoking and carbon monoxide poisoning, it becomes increasingly clear that this popular social activity carries significant health risks that extend far beyond those typically associated with to***co use.

يُمثل التسمم بأول أكسيد الكربون الناتج عن تدخين الشيشة خطرًا صحيًا جسيمًا، وإن كان غير مُدرك جيدًا. يُنتج الفحم المُستخدم لتسخين التبغ مستويات خطيرة من هذا الغاز عديم اللون والرائحة، والذي قد يُسبب مجموعة من الأعراض، تتراوح من الصداع والدوار إلى فقدان الوعي، وفي الحالات الشديدة، الوفاة. وقد ساهم الاعتقاد الخاطئ بأن ترشيح المياه يجعل تدخين الشيشة آمنًا في سلوكيات محفوفة بالمخاطر، لا سيما بين الشباب الذين يُمثلون الفئة السكانية الأكثر عرضة للانخراط في هذا النشاط. يُسلط الانتشار المتزايد لحالات التسمم بأول أكسيد الكربون المرتبطة بالشيشة في السنوات الأخيرة الضوء على الحاجة المُلحة لزيادة الوعي العام بهذا الخطر. ينبغي على مُقدمي الرعاية الصحية، ومسؤولي الصحة العامة، والمُعلمين العمل على نشر معلومات دقيقة حول مخاطر التعرض لأول أكسيد الكربون الناتج عن تدخين الشيشة. بالإضافة إلى ذلك، ينبغي على الأفراد الذين يختارون تدخين الشيشة اتخاذ الاحتياطات اللازمة لتقليل مخاطرهم من خلال ضمان التهوية الجيدة والانتباه لأعراض التسمم بأول أكسيد الكربون. ومع استمرار الأبحاث في توثيق العلاقة بين تدخين الشيشة والتسمم بأول أكسيد الكربون، أصبح من الواضح بشكل متزايد أن هذا النشاط الاجتماعي الشعبي يحمل مخاطر صحية كبيرة تمتد إلى ما هو أبعد من تلك المرتبطة عادة باستخدام التبغ.

27/02/2025

Pearl

Correction of Anemia by SGLT2 Inhibition in Patients with CKD and low EFHF;Recent studies
The erythrocytic effect of SGLT2 inhibitors has been evaluated in 5060 patients with CKD and anemia in four large-scale long-term trials–Two trials focused on patients with chronic heart failure (half of whom had CKD), and two trials focused on patients with CKD, with or without diabetes. In these four trials, SGLT2 inhibition increased hemoglobin and hematocrit after 4–12 weeks. Approximately 50%–70% of treated patients achieved hemoglobin and hematocrit levels in the nonanemic range.

20/02/2025

How do ventilation-perfusion mismatching and minute ventilation changes contribute to oxygen-induced hypercapnia in COPD

In COPD patients, ventilation-perfusion (V/Q) mismatching and changes in minute ventilation are key factors in oxygen-induced hypercapnia.

Ventilation-Perfusion (V/Q) Mismatching:

• Mechanism: COPD exacerbations often lead to alterations in the V/Q ratio due to bronchospasm, edema, and secretions, causing some areas of the lungs to be perfused but not adequately ventilated.

• Hypoxic Pulmonary Vasoconstriction: In COPD, the body uses hypoxic pulmonary vasoconstriction to improve gas exchange by altering V/Q ratios. However, oxygen therapy can counteract this mechanism, increasing V/Q mismatch and contributing to hypercapnia. Supplemental oxygen can disrupt the balance in the lungs by increasing PaO2 in diseased sections and diverting perfusion away from better-functioning areas. This results in shunting and dead space ventilation, ultimately leading to hypercapnia.

• Increased Dead Space Ventilation: V/Q mismatch can increase dead space ventilation, reducing the efficiency of carbon dioxide removal.

Minute Ventilation Changes:

• Hypoventilation: Hypercapnia is mainly caused by alveolar hypoventilation. A reduction in minute ventilation can cause hypercapnia.

• Respiratory Drive: While it was traditionally thought that supplemental oxygen suppresses the respiratory drive in COPD patients, causing hypoventilation and CO2 retention, its role is less significant than previously believed. Some studies suggest that the drop in minute ventilation is transient and does not correlate with increasing PaCO2 levels.

• Increased Respiratory Work: Hypercapnia can occur when a patient cannot sustain adequate ventilation due to the respiratory system being overloaded.

23/01/2025

يمكن بدأ ال prophylactic LMWH للمرضي المصابين ب spontaneous ICH اذا كان فيه risk of DVT و ذلك بعد مرور 48 ساعه من حدوث ال ICH و ذلك بعد التاكد من استقرار النزيف عن طريق ال follow up CT.
اما بالنسبة لل subarachnoid haemorrhage فيمكن البدأ في ال pharmacological thromboprophylaxis بعد 24 ساعه من علاج ال aneurysm.

07/09/2024

Every 4 units of blood will require 10 mL of 10% calcium gluconate.

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