Dr Sharmeen Aslam

Dr Sharmeen Aslam 🌿 Internal Medicine Consultant | Health Simplified
💻 Online Consults | 🩺 Evidence-based Wellness
👇 Follow for daily health tips & myth-busting.

Your happiness doesn’t need permission, validation, or applause. It begins the moment you choose yourself, protect your ...
06/08/2026

Your happiness doesn’t need permission, validation, or applause. It begins the moment you choose yourself, protect your peace, and honor your own journey. 🌿✨

What are you doing today to choose your happiness?



SelfLove HealingJourney PersonalGrowth DailyReminder Happiness WellBeing MentalWellness GrowthMindset Inspiration Motivation PeaceOfMind LifeQuotes MindfulLiving QuoteOfTheDay InstagramQuotes

30/07/2026

Portal Vein Thrombosis (PVT): A Practical Guide

Portal vein thrombosis is the formation of a blood clot within the portal vein, reducing blood flow from the intestines, spleen, and pancreas to the liver. Although it may present with vague symptoms, delayed diagnosis can lead to serious complications such as intestinal ischemia, portal hypertension, and variceal bleeding.

In this , you’ll learn:
✔️ What portal vein thrombosis is
✔️ How to diagnose it
✔️ Evidence-based treatment and follow-up

Early Doppler ultrasound and contrast-enhanced CT or MR venography are key to diagnosis. In most patients without contraindications, prompt anticoagulation improves portal vein recanalization and reduces thrombus progression. Investigating the underlying cause, including thrombophilia, myeloproliferative disorders, malignancy, and local inflammatory conditions, is essential for long-term management.

💡 Key takeaway: Early recognition and timely anticoagulation can significantly improve outcomes.

⚠️ This post is for educational purposes only and does not replace professional medical advice.

🩺 Rumination Syndrome in Autism: What Every Parent Should KnowDoes your child bring food back into their mouth shortly a...
27/07/2026

🩺 Rumination Syndrome in Autism: What Every Parent Should Know
Does your child bring food back into their mouth shortly after eating without nausea or vomiting? It may be rumination syndrome, a treatable condition that is more common in children with autism than many people realize.

The good news is that treatment does not usually rely on medication. The most effective approach is diaphragmatic breathing, combined with behavioral therapy, structured mealtime routines, and family support. With early recognition and consistent practice, many children experience significant improvement.

Remember:
✔️ It is not the child’s fault.
✔️ It is not the same as vomiting or acid reflux.
✔️ Early intervention leads to better outcomes.

💙 Every child deserves understanding, patience, and the right support.

Save this post to help raise awareness and share it with parents, caregivers, teachers, and healthcare professionals who may benefit.

20/07/2026

👁️ Holmes-Adie Pupil (Adie’s Tonic Pupil)

A dilated pupil that reacts poorly to light but constricts better with near focus is the hallmark of Holmes-Adie pupil. It results from damage to the ciliary ganglion or short ciliary nerves, leading to a tonic pupil with light-near dissociation.

💡 High-yield facts:
• Usually affects young adults, especially women
• Typically unilateral at presentation
• Slow redilation after near response (“tonic” pupil)
• Associated with absent or reduced Achilles tendon reflex in Holmes-Adie syndrome
• Dilute pilocarpine (0.125%) causes constriction due to denervation supersensitivity

📚 MRCP Pearl:
Large pupil + poor light response + better near response + positive dilute pilocarpine test = Holmes-Adie syndrome.

Save this post for your neurology revision and share it with your study partner!

18/07/2026

👁️ Argyll Robertson Pupil: A classic example of light-near dissociation.

🧠 Key finding:
❌ No constriction to light
✅ Constriction with accommodation (near response)

💡 Remember:
“Accommodates but does not react.”

This distinctive pupillary abnormality is classically associated with neurosyphilis (tabes dorsalis) and is a favorite topic in MBBS, FCPS, MRCP, USMLE, and NEET-PG examinations.

Save this post for quick revision and share it with your study partner!

15/07/2026

🧠 Crossed vs Uncrossed Hemiplegia: A Brain Localization Trick You Should Never Forget!

When examining a patient with hemiplegia, the pattern of weakness is often more important than the degree of weakness.

✅ Crossed Hemiplegia
• Ipsilateral cranial nerve palsy
• Contralateral body weakness
• 📍Localizes to the brainstem

✅ Uncrossed Hemiplegia
• Face, arm, and leg weakness on the same side
• 📍Localizes to the cerebral hemisphere or internal capsule (supratentorial lesion)

🎯 High-Yield MRCP Pearl

Crossed findings = Brainstem lesion 🧠

No crossed findings = Think supratentorial lesion

This simple concept helps localize lesions quickly in stroke, demyelinating disease, tumors, and other neurological disorders.

💬 Question: Which brainstem syndrome causes ipsilateral CN III palsy with contralateral hemiplegia? Answer in the comments!

📚 Save this post for MRCP, USMLE, FCPS, MD, NEET PG, PLAB, and Internal Medicine revision.

🧠 Neuroanatomy Challenge: Can You Localize the Lesion?A patient presents with right-sided LMN facial weakness (CN VII pa...
13/07/2026

🧠 Neuroanatomy Challenge: Can You Localize the Lesion?

A patient presents with right-sided LMN facial weakness (CN VII palsy) and left-sided hemiplegia.

📍 Where is the lesion?

A. Right internal capsule
B. Left internal capsule
C. Right pons
D. Left cerebral hemisphere

💬 Drop your answer (A, B, C, or D) in the comments before checking the next post!

This is a classic crossed neurological deficit that frequently appears in MRCP, USMLE, PLAB, FCPS, and NEET PG exams. Mastering these localization patterns makes brainstem lesions much easier to identify in clinical practice.

📌 Save this post for revision.
🔄 Share it with your study partner.
❤️ Follow for high-yield Internal Medicine and Neurology MCQs.

Hemiplegia

10/07/2026

👀 Conjugate Eye Movement Pathway Made Simple!

Understanding conjugate gaze is much easier when you know the roles of the Frontal Eye Field (FEF), PPRF, MLF, Abducens nucleus, and Oculomotor nucleus.

🧠 High-yield clinical pearls:
• FEF initiates voluntary saccades to the opposite side.
• PPRF is the horizontal gaze center.
• MLF coordinates both eyes for conjugate horizontal gaze.
• PPRF lesion → Ipsilateral horizontal gaze palsy.
• Abducens nucleus lesion → Ipsilateral horizontal gaze palsy.
• Abducens nerve lesion → Isolated ipsilateral lateral re**us palsy (not horizontal gaze palsy).
• MLF lesion causes Internuclear Ophthalmoplegia (INO) with impaired adduction and abducting nystagmus.

Save this post for your next neurology revision and share it with friends preparing for MRCP, USMLE, NEET PG, FCPS, or Internal Medicine.

09/07/2026

🧠 MND (ALS) Updated Treatment Guidelines

Managing Motor Neuron Disease goes beyond prescribing medication. Early diagnosis, timely disease-modifying therapy, respiratory monitoring, nutritional support, and multidisciplinary care all play a vital role in improving survival and quality of life.

Key Takeaways

✅ Riluzole remains the first-line disease-modifying therapy for most patients.

✅ Edaravone may be considered in selected patients with early disease.

✅ Monitor respiratory function regularly and initiate non-invasive ventilation (BiPAP) when indicated.

✅ Address nutrition early and consider PEG feeding before advanced respiratory compromise.

✅ Treat symptoms proactively, including spasticity, sialorrhea, pseudobulbar affect, and muscle cramps.

💡 High-Yield Pearl:
Early referral to a multidisciplinary ALS clinic is one of the most effective interventions for improving both survival and quality of life.

📌 Save this post for revision and share it with colleagues preparing for MRCP, FCPS, USMLE, PLAB, or NEET-PG.

08/07/2026

✅ Correct Answer: B. Motor Neuron Disease (MND/ALS)

The key clues were:
• Progressive asymmetric limb weakness
• Fasciculations (LMN involvement)
• Normal MRI excluding structural lesions
• EMG showing widespread denervation

These findings strongly point toward Motor Neuron Disease (ALS).

💡 Exam Pearl:
Progressive weakness + fasciculations + normal sensation + normal MRI + widespread denervation on EMG = Think MND/ALS.

Save this post for revision and share it with your study partner!

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