Yggdrasil Naturopathic

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🌱Naturopathic Medicine
🌿Herbalism
🙏Mindbody
🐲Acupuncture & Chinese Medicine
🥑Nutrition & Exercise Science🏋️

📲 952.295.4036
14525 Highway 7, Suite 375, Minnetonka, Minnesota 55345
linktr.ee/yggnatmed
https://wayfinderswell.com

10/20/2026

Standing up is a small circulatory event. Gravity pulls blood into the lower body, and the system has to push it back up so the brain and other organs stay supplied.

There are a few ways that compensation can fail, and the differences between them are what separate these diagnoses.
In POTS, the heart rate goes up and stays up. The usual reason is that blood vessels in the lower body aren’t constricting the way they should, so blood pools there, and the heart compensates by beating faster to move it upward.

The detail that distinguishes POTS is blood pressure. It often doesn’t change at all, and in some cases, it rises slightly. The tachycardia is doing the work of holding perfusion steady.

Orthostatic hypotension looks different on the cuff. Standing up produces a blood pressure drop.

The underlying causes can overlap considerably, which is why some of the treatments do too. Both can involve lower-body blood vessels that fail to compensate, and both can involve inadequate fluid volume in the system.

Same trigger, different failure, different numbers on standing.

10/19/2026

A pathology report listing eosinophils or mast cells reads differently depending on which cells are named, and the difference isn’t about the cells being good or bad.

Eosinophils are normal cells with a job, usually tied to allergic and inflammatory responses. High density is the signal, not presence on its own. Eosinophilic esophagitis makes this concrete. Healthy esophageal mucosa contains no eosinophils at all, so any infiltration there means something is actively driving them in.

Mast cells live in tissue by design. They sit in the surface layers of every barrier where the body meets the outside world, which is the skin, gut, and the respiratory tract. They’re stationed there as sentinels. Seeing them in those places is what you’d expect.
The abnormal finding with mast cells is clumping. Too many people packed into one area is what flags a problem.

So two different questions get asked of the same report. Are eosinophils somewhere they shouldn’t be? Are mast cells distributed the way they should be?

10/16/2026

Roughly three out of four people with POTS had a doctor suggest their symptoms were psychiatric or psychological before anyone said POTS. Only about 28% of them reported actually having a psychiatric problem at the time.

The cost of that is measured in years. About one in seven people waited more than 10 years for a POTS diagnosis.

What makes the anxiety framing wrong is the order in which things happen. In POTS, the cardiovascular dysfunction comes first. The heart races because the autonomic nervous system isn’t keeping blood in the upper body on standing. The racing heart, the tremors, the sense that something bad is about to happen, all of it follows from that physiological failure rather than causing it.

There’s imaging to go with this. The same Dallas research group put people in an MRI and found the heart muscle about 16% smaller by mass in POTS patients compared to matched sedentary healthy controls. Lower blood volume, smaller heart muscle, and reduced peak oxygen uptake aren’t things anxiety produces.

And many people spend months waiting on a tilt table test that may not even be necessary.

10/15/2026

The low histamine diet is hard to follow partly because nobody can tell you what’s actually on it.

A 2021 study in Nutrients reviewed 10 published low-histamine diets and found that none of them agreed with each other. Fermented foods were the only thing on every single list. Cured foods made many of them. Past that, the lists were a scattering of items that may or may not contain much histamine at all.
The reason they diverge is that foods land on these lists for different reasons, and those reasons go unlabeled.

A lot of what appears isn’t high in histamine. Those foods are histamine liberators, meaning they appear to irritate mast cells and eosinophils into dumping histamine. The issue isn’t how much histamine is in the food. It’s how the body responds to it.
Whether any given reason applies to you is a separate question, and that’s exactly why labeling matters. Without it, everyone eliminates everything.

10/14/2026

Iron dosing has shifted considerably over the past decade, and the reason comes down to hepcidin.

Every iron dose raises hepcidin. Hepcidin downregulates ferroportin, which is what moves iron out of intestinal cells and into circulation. So each dose reduces how well the next one gets absorbed.

The timing matters. Hepcidin peaks around eight hours after a dose and is still elevated at 24 hours. It doesn’t come back down until about 48 hours. Which means a dose taken the following morning is absorbed roughly 35 to 45% less effectively than it would have been otherwise.

That’s the case for alternate-day dosing. A single morning dose every other day, taken with vitamin C and away from coffee, is a reasonable default for routine repletion. The same total iron produces the same ferritin, with fewer gut symptoms, and it’s a regimen people can actually stay on.

One more thing worth saying. People tend to stop once they feel better, and that’s usually too early. Feeling better and being replete aren’t the same thing, so the labs are what should decide when to stop.

10/14/2026

Mast cells showing up on a biopsy isn’t a problem on its own. What marks it as abnormal is clumping. Too many mast cells concentrated in one area is what signals that something is going on.

Eosinophilic disorders follow a different treatment path entirely. They’re handled along allergy lines, with corticosteroids and certain inflammatory marker inhibitors doing most of the work.

That split is the practical reason the distinction between these two cell types matters. The treatments aimed at eosinophilia are built for a different mechanism than the ones aimed at mast cell problems. Applying one to the other doesn’t produce the same result, which is why the pathology report has to be read carefully before a protocol is chosen.

10/13/2026

Eosinophils are the cells most often associated with allergic disease, and when they appear in high density on a biopsy, they’re pointing toward an allergic or inflammatory process.

Worth clarifying something, though. Eosinophils aren’t abnormal cells. They’re normal cells with a specific job to do. Finding them isn’t the finding. Finding them in high numbers is because that density says something is driving them there.

This is also why matching treatment to the right cell line matters. A protocol built for eosinophilic disease applied to a mast cell problem won’t do much, and mast cell treatments applied to an eosinophil-driven process won’t either. The result in both cases is the same: no relief, or symptoms that keep progressing while the wrong approach gets a trial.

Which cells are elevated is what points toward the right treatment.

10/12/2026

Mast cells turning up on a biopsy is one thing. Mast cells turning up in large numbers, in clumps, is a different situation.

That picture prompts a workup for clonal disease, including KIT mutation mastocytosis. The point of that workup is ruling things out.

Clonal means overproliferation. Too many copies of one cell type. And whenever clonality comes up in medicine, the concern sitting behind it is cancer, because uncontrolled replication of a single cell line is what cancer is.

So when a biopsy shows mast cells in massive numbers, the question is whether the body is producing them in an abnormal, uncontrolled way. That isn’t normal behavior for mast cells, and it’s worth investigating properly rather than assuming it’s the same mast cell activation picture seen in MCAS.

Most of the time, that workup comes back clear. Doing it anyway is what makes the clear result meaningful.

10/09/2026

Both mast cells and eosinophils produce histamine, which is part of why they get conflated. But when either one turns up on a biopsy, the reason it’s there can be very different.

That difference carries through to everything downstream. The two cell lines are associated with different diseases, which means different diagnoses, which means different treatments.

This is the whole function of getting the diagnosis right. A treatment aimed at eosinophilic disease applied to a mast cell problem won’t do much. The same is true in reverse. Mast cell stabilizers and antihistamines aimed at what’s actually an eosinophil-driven process won’t deliver the relief someone is looking for.

The practical cost of getting that wrong is time. Symptoms either stay where they are or keep progressing while a protocol that was never going to fit gets tried out.

So when a biopsy report comes back with elevated cells, which cells they are matters for what comes next.

10/09/2026

The bitter herbs used for small intestinal motility work in part by stimulating bile flow, and the reason that helps comes down to what bile actually does in the gut.

Bile is a signaling molecule. When it reaches the first part of the small intestine, it puts the brake on the last part. That braking is what creates the coordinated wave that moves contents forward in sequence rather than everything contracting at once.

So bile isn't only about digesting fats. It's part of how the small intestine organizes its movement.

Which is why bile keeps coming up in these conversations. You can't simply remove it from the equation. Even without a gallbladder, the liver continues producing bile and dripping it into the small intestine, and that signaling continues doing its job.

For anyone working on motility, supporting bile flow is working on the signal that coordinates the movement in the first place.

Address

14525 Highway 7, Suite 375
Minnetonka, MN
55345

Opening Hours

Tuesday 10:30am - 4pm
Wednesday 10:30am - 7pm
Thursday 10:30am - 7pm
Friday 10:30am - 7pm
Saturday 10:30am - 7pm

Telephone

+13203487424

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